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uacc 3199  (ATCC)


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    ATCC uacc 3199
    ( A ). Cell viability of cells treated with curcumin for 24 h. Viability was determined by the resazurin assay. Data were averaged from three independent experiments with six replicas each, and the standard deviation is shown. ( B) . VEGF concentrations in cell culture supernatant upon 24 h exposure to IL-1β, curcumin, and combinations of both were determined by ELISA for ( B ) MCF-7, ( C ) SK-BR-3, ( D ) MDA-MB-231, and ( E ) <t>UACC-3199.</t> Data are averaged from three independent experiments with two replicas each, and median and quartiles are shown. ( F) . Effects of inhibition of MAP-kinases on curcumin-induced VEGF secretion in MCF-7. SB: SB203580 (p38-MAPK-inhibitor); UO: UO126 (ERK-inhibitor). Data are expressed relative to solvent-treated cells. Data averaged from three independent experiments with three replicas each, and the standard error is shown. * p < 0.05, n.s.: not significant (ANOVA).
    Uacc 3199, supplied by ATCC, used in various techniques. Bioz Stars score: 93/100, based on 31 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/uacc+3199/pmc12963662-114-10-11?v=ATCC
    Average 93 stars, based on 31 article reviews
    uacc 3199 - by Bioz Stars, 2026-07
    93/100 stars

    Images

    1) Product Images from "Interplay of Interleukin-1 β and Curcumin on VEGF Expression in Breast Cancer Cells"

    Article Title: Interplay of Interleukin-1 β and Curcumin on VEGF Expression in Breast Cancer Cells

    Journal: Oncology Research

    doi: 10.32604/or.2025.072793

    ( A ). Cell viability of cells treated with curcumin for 24 h. Viability was determined by the resazurin assay. Data were averaged from three independent experiments with six replicas each, and the standard deviation is shown. ( B) . VEGF concentrations in cell culture supernatant upon 24 h exposure to IL-1β, curcumin, and combinations of both were determined by ELISA for ( B ) MCF-7, ( C ) SK-BR-3, ( D ) MDA-MB-231, and ( E ) UACC-3199. Data are averaged from three independent experiments with two replicas each, and median and quartiles are shown. ( F) . Effects of inhibition of MAP-kinases on curcumin-induced VEGF secretion in MCF-7. SB: SB203580 (p38-MAPK-inhibitor); UO: UO126 (ERK-inhibitor). Data are expressed relative to solvent-treated cells. Data averaged from three independent experiments with three replicas each, and the standard error is shown. * p < 0.05, n.s.: not significant (ANOVA).
    Figure Legend Snippet: ( A ). Cell viability of cells treated with curcumin for 24 h. Viability was determined by the resazurin assay. Data were averaged from three independent experiments with six replicas each, and the standard deviation is shown. ( B) . VEGF concentrations in cell culture supernatant upon 24 h exposure to IL-1β, curcumin, and combinations of both were determined by ELISA for ( B ) MCF-7, ( C ) SK-BR-3, ( D ) MDA-MB-231, and ( E ) UACC-3199. Data are averaged from three independent experiments with two replicas each, and median and quartiles are shown. ( F) . Effects of inhibition of MAP-kinases on curcumin-induced VEGF secretion in MCF-7. SB: SB203580 (p38-MAPK-inhibitor); UO: UO126 (ERK-inhibitor). Data are expressed relative to solvent-treated cells. Data averaged from three independent experiments with three replicas each, and the standard error is shown. * p < 0.05, n.s.: not significant (ANOVA).

    Techniques Used: Resazurin Assay, Standard Deviation, Cell Culture, Enzyme-linked Immunosorbent Assay, Inhibition, Solvent



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    ( A ). Cell viability of cells treated with curcumin for 24 h. Viability was determined by the resazurin assay. Data were averaged from three independent experiments with six replicas each, and the standard deviation is shown. ( B) . VEGF concentrations in cell culture supernatant upon 24 h exposure to IL-1β, curcumin, and combinations of both were determined by ELISA for ( B ) MCF-7, ( C ) SK-BR-3, ( D ) MDA-MB-231, and ( E ) <t>UACC-3199.</t> Data are averaged from three independent experiments with two replicas each, and median and quartiles are shown. ( F) . Effects of inhibition of MAP-kinases on curcumin-induced VEGF secretion in MCF-7. SB: SB203580 (p38-MAPK-inhibitor); UO: UO126 (ERK-inhibitor). Data are expressed relative to solvent-treated cells. Data averaged from three independent experiments with three replicas each, and the standard error is shown. * p < 0.05, n.s.: not significant (ANOVA).
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    ( A ). Cell viability of cells treated with curcumin for 24 h. Viability was determined by the resazurin assay. Data were averaged from three independent experiments with six replicas each, and the standard deviation is shown. ( B) . VEGF concentrations in cell culture supernatant upon 24 h exposure to IL-1β, curcumin, and combinations of both were determined by ELISA for ( B ) MCF-7, ( C ) SK-BR-3, ( D ) MDA-MB-231, and ( E ) <t>UACC-3199.</t> Data are averaged from three independent experiments with two replicas each, and median and quartiles are shown. ( F) . Effects of inhibition of MAP-kinases on curcumin-induced VEGF secretion in MCF-7. SB: SB203580 (p38-MAPK-inhibitor); UO: UO126 (ERK-inhibitor). Data are expressed relative to solvent-treated cells. Data averaged from three independent experiments with three replicas each, and the standard error is shown. * p < 0.05, n.s.: not significant (ANOVA).
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    ( A ). Cell viability of cells treated with curcumin for 24 h. Viability was determined by the resazurin assay. Data were averaged from three independent experiments with six replicas each, and the standard deviation is shown. ( B) . VEGF concentrations in cell culture supernatant upon 24 h exposure to IL-1β, curcumin, and combinations of both were determined by ELISA for ( B ) MCF-7, ( C ) SK-BR-3, ( D ) MDA-MB-231, and ( E ) <t>UACC-3199.</t> Data are averaged from three independent experiments with two replicas each, and median and quartiles are shown. ( F) . Effects of inhibition of MAP-kinases on curcumin-induced VEGF secretion in MCF-7. SB: SB203580 (p38-MAPK-inhibitor); UO: UO126 (ERK-inhibitor). Data are expressed relative to solvent-treated cells. Data averaged from three independent experiments with three replicas each, and the standard error is shown. * p < 0.05, n.s.: not significant (ANOVA).
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    Image Search Results


    ( A ). Cell viability of cells treated with curcumin for 24 h. Viability was determined by the resazurin assay. Data were averaged from three independent experiments with six replicas each, and the standard deviation is shown. ( B) . VEGF concentrations in cell culture supernatant upon 24 h exposure to IL-1β, curcumin, and combinations of both were determined by ELISA for ( B ) MCF-7, ( C ) SK-BR-3, ( D ) MDA-MB-231, and ( E ) UACC-3199. Data are averaged from three independent experiments with two replicas each, and median and quartiles are shown. ( F) . Effects of inhibition of MAP-kinases on curcumin-induced VEGF secretion in MCF-7. SB: SB203580 (p38-MAPK-inhibitor); UO: UO126 (ERK-inhibitor). Data are expressed relative to solvent-treated cells. Data averaged from three independent experiments with three replicas each, and the standard error is shown. * p < 0.05, n.s.: not significant (ANOVA).

    Journal: Oncology Research

    Article Title: Interplay of Interleukin-1 β and Curcumin on VEGF Expression in Breast Cancer Cells

    doi: 10.32604/or.2025.072793

    Figure Lengend Snippet: ( A ). Cell viability of cells treated with curcumin for 24 h. Viability was determined by the resazurin assay. Data were averaged from three independent experiments with six replicas each, and the standard deviation is shown. ( B) . VEGF concentrations in cell culture supernatant upon 24 h exposure to IL-1β, curcumin, and combinations of both were determined by ELISA for ( B ) MCF-7, ( C ) SK-BR-3, ( D ) MDA-MB-231, and ( E ) UACC-3199. Data are averaged from three independent experiments with two replicas each, and median and quartiles are shown. ( F) . Effects of inhibition of MAP-kinases on curcumin-induced VEGF secretion in MCF-7. SB: SB203580 (p38-MAPK-inhibitor); UO: UO126 (ERK-inhibitor). Data are expressed relative to solvent-treated cells. Data averaged from three independent experiments with three replicas each, and the standard error is shown. * p < 0.05, n.s.: not significant (ANOVA).

    Article Snippet: All cell lines MCF-7 (ATCC-HTB-22), SK-BR-3 (ATCC-HTB-30), MDA-MB-231 (ATCC-HTB-26), and UACC-3199 (ATCC-CRL-2983) were obtained from the American Type Culture Collection (ATCC, via LGC Standards, Wesel, Germany).

    Techniques: Resazurin Assay, Standard Deviation, Cell Culture, Enzyme-linked Immunosorbent Assay, Inhibition, Solvent

    Expression of LINC00992 in breast cancer cell lines determined by quantitative reverse transcription polymerase chain reaction

    Journal: Contemporary Oncology

    Article Title: The tamoxifen-regulated, long non-coding RNA LINC00992 affects proliferation, migration, and expression of tamoxifen resistance-associated genes in MCF-7 breast cancer cells

    doi: 10.5114/wo.2023.125000

    Figure Lengend Snippet: Expression of LINC00992 in breast cancer cell lines determined by quantitative reverse transcription polymerase chain reaction

    Article Snippet: MCF-7, T47D, UACC3199, HS578T, MDA-MB468, SkBr3, and MDA-MB-231 cells were obtained from the American Type Culture Collection (ATCC ® , Manassas, VA, USA).

    Techniques: Expressing, Reverse Transcription, Standard Deviation